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Human Bronchial Epithelial Cells: HBEpC, adult

ITEM#: 3042-50205A

MFR#: 502-05A

HBEpC provide an excellent model system to study all aspects of epithelial function and disease, particularly those related to airway viral infections, as well as tissue repair mechanisms, signaling changes and potential treatments relevant to lung i

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HBEpC provide an excellent model system to study all aspects of epithelial function and disease, particularly those related to airway viral infections, as well as tissue repair mechanisms, signaling changes and potential treatments relevant to lung injuries, mechanical and oxidative stress, inflammation, pulmonary diseases and smoking. When grown on inserts and provided with the liquid/air interface, HBEpC can differentiate into a pseudostriated epithelium and serve as a more physiological 3D tissue model for in vitro studies.
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Product Name Description Item# QTY
Human Bronchial Epithelial Cells (HBEpC) 500,000 cryopreserved cells Human Bronchial Epithelial Cells (HBEpC) 500,000 cryopreserved cells 3042-C12640 EA
Human Bronchial Epithelial Cells (HBEpC) 1 million cells per pellet Human Bronchial Epithelial Cells (HBEpC) 1 million cells per pellet 3042-C14063 EA
16HBE14o-Human Bronchial Epithelial Cell Line 16HBE14o- is a human bronchial epithelial cell line isolated from a 1-year old male heart-lung patient and immortalized with the origin-of-replication defective SV40 plasmid (pSVori-). The cell line retains characteristic features of normal different 3042-SCC150 EA
CFBE41o- Human CF Bronchial Epithelial Cell Line CFBE41o- human CF bronchial epithelial cell line was derived from a cystic fibrosis patient homozygous for the DeltaF508 CFTR mutation. Cystic Fibrosis (CF) is a lethal autosomal recessive disease caused by mutations in the CF transmembrane conductance regulator (CFTR) gene which functions as a cAMP-activated and phosphorylated-regulated Cl channel. The predominant mutation in the CF 3042-SCC151 EA
CFBE41o- 4.7 WT-CFTR Human CF Bronchial Epithelial Cystic Fibrosis (CF) is a lethal autosomal recessive disease caused by mutations in the CF transmembrane conductance regulator (CFTR) gene which functions as a cAMP-activated and phosphorylated-regulated Cl channel. The predominant mutation in the CF 3042-SCC158 EA
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