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Bovine Coronary Artery Smooth Muscle Cells: BCASMC (Cryovial)

ITEM#: 3042-B35005

MFR#: B350-05

BCASMC have been used to test novel photocrosslinked biodegradable elastomers based material surfaces for their usefulness in smooth muscle tissue engineering (Ilagan, 2009).Characterization: positive for smooth muscle cell specific alpha-actin expre

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BCASMC have been used to test novel photocrosslinked biodegradable elastomers based material surfaces for their usefulness in smooth muscle tissue engineering (Ilagan, 2009).Characterization: positive for smooth muscle cell specific alpha-actin expression.
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Product Name Description Item# QTY
Porcine Coronary Artery Smooth Muscle Cells: PCASMC (Cryovial) PCASMC were used to investigate the role of c-Jun in smooth muscle proliferation, and to show that c-Jun inhibition abrogates smooth muscle cell growth and intimal thickening after arterial injury (Khachigian, 2002). PCASMC were also used to develop 3042-P35005 EA
Canine Coronary Artery Smooth Muscle Cells: CnCASMC (Cryovial) Canine Coronary Artery Smooth Muscle Cells (CnCASMC) provide an excellent animal model system to study all aspects of cardiovascular function and disease. 3042-CN35005 EA
Bovine Renal Artery Smooth Muscle Cells: BRASMC (Cryovial) Bovine Renal Artery Smooth Muscle Cells (BRASMC) are derived from tunica intima and tunica media of normal bovine renal arteries. They are cryopreserved at second passage and can be cultured and propagated at least 12 population doublings. Studies sh 3042-B95005 EA
Bovine Pulmonary Artery Smooth Muscle Cells: BPASMC (Cryovial) BPASMC have been used in studies relevant to pulmonary hypertension characterized by hyperproliferation of pulmonary artery smooth muscle cells, the process shown to be mediated by hypoxia-induced expression of Bcl-xl (Suzuki, 2007). Additionally, th 3042-B35205 EA
Bovine Coronary Artery Endothelial Cells: BCAEC (Cryovial) BCAEC have been used to demonstrate that the stress hormone cortisol inhibits NO synthase and Ca2+ mobilization leading to decreased NO release by endothelial cells, resulting in increased arterial contraction and decreased blood flow (Rogers, 2002). 3042-B30005 EA
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